Xiao-Jun Du | Biochemistry | Best Review Article Award

Best Researcher Award
Xiao-Jun Du — Baker Heart and Diabetes Institute, Australia
Xiao-Jun Du
Affiliation Baker Heart and Diabetes Institute
Country Australia
Scopus ID 7402551956
Documents 241
Citations 12,313
h-index 62
Subject Area Biochemistry
Event Best Paper Awards

Xiao-Jun Du is a researcher associated with the Baker Heart and Diabetes Institute, Australia, whose work contributes to the field of Biochemistry with emphasis on metabolic regulation and cardiovascular disease mechanisms. This article summarizes his academic profile, research output, and scholarly impact using bibliometric indicators such as Scopus records, citation metrics, and h-index evaluation. With 241 documents and 12,313 citations, his research demonstrates consistent contributions to biomedical science. The profile also discusses recognition through awards including the Best Paper Awards event. Overall, the study highlights his influence in translational research and potential suitability for scientific recognition and academic leadership globally.

Abstract

Xiao-Jun Du is a researcher associated with the Baker Heart and Diabetes Institute, Australia, whose work contributes to the field of Biochemistry with emphasis on metabolic regulation and cardiovascular disease mechanisms. This article summarizes his academic profile, research output, and scholarly impact using bibliometric indicators such as Scopus records, citation metrics, and h-index evaluation. With 241 documents and 12,313 citations, his research demonstrates consistent contributions to biomedical science. The profile also discusses recognition through awards including the Best Paper Awards event. Overall, the study highlights his influence in translational research and potential suitability for scientific recognition and academic leadership globally.

Keywords

Biochemistry; Cardiovascular Disease; Metabolic Regulation; Bibliometrics; Scopus; h-index; Translational Research; Biomedical Science

Introduction

Xiao-Jun Du is an academic researcher affiliated with the Baker Heart and Diabetes Institute, Australia, working in Biochemistry and related biomedical sciences. His research focuses on molecular mechanisms underlying cardiometabolic diseases. This article presents an overview of his scholarly metrics, publications, and academic recognition within global scientific communities worldwide impact.

Research Profile

Xiao-Jun Du’s research profile reflects extensive contributions to cardiovascular and metabolic biochemistry. He has authored numerous peer-reviewed publications and collaborated internationally. His academic output is indexed in Scopus, demonstrating sustained research productivity. The profile highlights his expertise in disease mechanisms, biomarker discovery, and translational biomedical research applications and innovation focus.

Research Contributions

His research contributions include significant advancements in understanding cardiovascular disease pathways and metabolic regulation. He has contributed to experimental studies identifying molecular targets for therapy development. His interdisciplinary work bridges basic science and clinical research, strengthening translational outcomes and supporting innovation in biomedical diagnostics and treatment strategies global research impact.

Publications

His publication record comprises articles in peer-reviewed international journals focusing on biochemistry, cardiometabolic disease, and molecular biology. Indexed databases such as Scopus reflect his consistent scholarly output. These publications contribute to advancing scientific understanding and support ongoing research collaborations across global academic institutions and interdisciplinary biomedical knowledge and expansion globally.

Research Impact

His research impact is evidenced by citation metrics, academic recognition, and contributions to high-impact biomedical studies. With over 12,313 citations and an h-index of 62, his work demonstrates strong influence in the scientific community. His findings support advancements in cardiovascular and metabolic disease research worldwide and translational science relevance globally.

Award Suitability

His academic profile indicates strong suitability for recognition in international scientific awards. His contributions to biochemistry and cardiovascular research demonstrate consistent excellence and innovation. The combination of high citation impact, extensive publications, and interdisciplinary collaboration positions him as a candidate for distinguished research honors and professional acknowledgment global academic recognition.

Conclusion

This overview highlights Xiao-Jun Du’s contributions to biochemistry and biomedical research, emphasizing his academic productivity and citation impact. His work continues to support advancements in cardiovascular and metabolic disease understanding. Ongoing research engagement ensures sustained relevance in global scientific discourse and future biomedical innovation and continued academic research excellence globally.

External Links

References

  1. Elsevier. (n.d.). Scopus author details: Xiao-Jun Du, Author ID 7402551956. Scopus.
    https://www.scopus.com/authid/detail.uri?authorId=7402551956
  2. Best Paper Awards. (n.d.). Official event information and recognition listing.
    https://bestpaperawards.com/
  3. Galectin-3 gene deletion alters lipid profile in healthy and cardiomyopathy mouse hearts.
    https://www.researchgate.net/publication/394312305_Galectin-3_gene_deletion_alters_lipid_profile_in_healthy_and_cardiomyopathy_mouse_hearts

John Byrnes | Biochemistry, Genetics and Molecular Biology | Best Paper Award

Best Paper Award

Selective Inhibition of Proofreading Exonucleases The Central Role in Obesity Assoviated Carciniogenesis
John Byrnes
Affiliation University of Miami
Country United States
Article Title Selective Inhibition of Proofreading Exonucleases The Central Role in Obesity Assoviated Carciniogenesis
Google Scholar ID 1kizaHgAAAAJ&hl
Article Type Research Article
Article Views 870
Reference Count 88
Award Category Best Paper Award
Event International Research Excellence and Best Paper Awards
ORCID 0000-0003-0940-9710

The Best Paper Award recognizes the scholarly contribution of John Byrnes of the University of Miami for research examining the relationship between proofreading exonucleases and obesity-associated carcinogenesis. Published in MDPI during 2026, the study contributes to ongoing discussions in biochemistry, genetics, and molecular biology regarding genomic integrity, cancer development, and molecular mechanisms associated with metabolic disorders. The award acknowledges the scientific significance, methodological rigor, and interdisciplinary relevance of the publication within contemporary biomedical research.[1]

Abstract

This award-recognized research investigates the potential role of proofreading exonucleases in mechanisms associated with obesity-related carcinogenesis. The study explores how alterations in DNA replication fidelity, repair pathways, and genomic maintenance may contribute to cellular transformation under metabolic stress conditions. Through molecular and biochemical analyses, the research evaluates the consequences of selective exonuclease inhibition and its implications for tumor development. Findings provide insights into interactions between obesity-associated biological environments and genomic instability. The work advances understanding of cancer biology while identifying potential molecular targets for future therapeutic development and translational biomedical investigations.[2]

Keywords

AMP; AMPK; carcinogenesis; DNA polymerase; fidelity; energy regulation; metabolism; mutation; obesity; proofreading exonuclease.

Introduction

The increasing prevalence of obesity has intensified scientific interest in understanding its relationship with cancer risk and progression. Researchers have identified multiple biological pathways linking metabolic dysregulation to genomic instability. This study focuses on proofreading exonucleases, enzymes responsible for maintaining DNA replication accuracy, and evaluates their relevance within obesity-associated carcinogenic processes that may influence disease initiation and progression.[2]

Research Profile

John Byrnes is affiliated with the University of Miami and has established a research portfolio within biochemistry, genetics, and molecular biology. With 138 indexed scholarly documents, 8,573 citations, and an h-index of 39, his academic contributions demonstrate sustained engagement in molecular mechanisms underlying human disease, cancer development, and genomic maintenance processes across diverse biomedical research domains.[3]

Scientific Background

Proofreading exonucleases play a critical role in correcting replication errors and preserving genome integrity. Deficiencies in these enzymatic systems can result in elevated mutation rates and increased susceptibility to malignant transformation. Previous investigations have linked obesity-related inflammation, oxidative stress, and metabolic disturbances with DNA damage, creating a scientific basis for examining interactions between exonuclease activity and carcinogenic pathways.[4]

Methodology

The research applies molecular biology and biochemical methodologies to investigate the consequences of selective proofreading exonuclease inhibition. Experimental analyses assess cellular responses, genomic integrity markers, and mechanisms associated with DNA repair regulation. By integrating laboratory observations with established cancer biology frameworks, the study evaluates how altered proofreading functions may contribute to carcinogenic processes under obesity-associated physiological conditions.[2]

Key Findings

The study identifies significant relationships between proofreading exonuclease activity and mechanisms that influence genomic stability. Results indicate that disruptions in proofreading functions may enhance mutation accumulation and contribute to cellular environments favorable to carcinogenesis. The findings support the hypothesis that obesity-associated biological stressors can interact with genomic maintenance pathways, potentially increasing cancer susceptibility through complex molecular mechanisms.[2]

Scientific Contributions

This research contributes to the growing body of knowledge connecting metabolic disease and cancer biology. By highlighting proofreading exonucleases as important molecular components within obesity-associated carcinogenesis, the study offers a framework for future investigations into diagnostic biomarkers and therapeutic interventions. The work also strengthens interdisciplinary connections between molecular genetics, oncology, and translational biomedical science.[4]

Conclusion

The Best Paper Award recognizes a publication that advances understanding of molecular mechanisms underlying obesity-associated cancer development. Through examination of proofreading exonucleases and genomic stability pathways, the research provides valuable scientific insights with potential relevance to future therapeutic strategies. Its contribution to contemporary biomedical literature reflects both methodological rigor and significance within the broader field of molecular oncology.[5]

References

  1. MDPI. (2026). Selective Inhibition of Proofreading Exonucleases The Central Role in Obesity Assoviated Carciniogenesis.
    https://www.mdpi.com/1467-3045/48/4/346
  2. DOI Reference. Cell Biochemistry and Function, Volume 48, Issue 4.
    https://doi.org/10.3390/cimb48040346
  3. Google Scholar. (n.d.). John Byrnes Author Profile.
    https://scholar.google.com/citations?user=1kizaHgAAAAJ&hl=en&oi=sra
  4. MDPI. (2026). Journal Information and Publication Metadata.
    https://www.mdpi.com/
  5. International Research Excellence and Best Paper Awards. (2026). Best Paper Award Recognition Program.
    https://bestpaperawards.com/

Guenther Witzany | Biochemistry, Genetics and Molecular Biology | Excellence in Research Award

Excellence in Research Award

Guenther Witzany
Telos – Philosophische Praxis

Guenther Witzany
Researcher Guenther Witzany
Affiliation Telos – Philosophische Praxis
Country Austria
Scopus ID 14629725000
Documents 29
Citations 288
h-index 11
Subject Area Biochemistry, Genetics and Molecular Biology
Event International Research Excellence and Best Paper Awards

Guenther Witzany, affiliated with Telos – Philosophische Praxis, Austria, is recognized through the Excellence in Research Award for his scholarly contributions to biocommunication theory, evolutionary biology, genetics, and molecular biology. His interdisciplinary work explores communication processes within living systems and advances understanding of biological information exchange. The recognition reflects a sustained publication record, measurable citation impact, and contributions to contemporary scientific discourse within the broader field of molecular and evolutionary sciences.[1]

Abstract

The Excellence in Research Award acknowledges the academic contributions of Guenther Witzany in advancing interdisciplinary understanding of communication processes in living systems. His work integrates concepts from molecular biology, genetics, evolution, and philosophy of science to examine how biological entities generate, exchange, and interpret information. Through publications, theoretical frameworks, and scholarly engagement, Witzany has contributed to discussions concerning biological signaling, genetic regulation, and evolutionary adaptation. His research profile demonstrates sustained productivity, international visibility, and measurable scientific influence, supporting recognition within the International Research Excellence and Best Paper Awards program.[2]

Keywords

Biocommunication, Molecular Biology, Evolutionary Theory, Genetics, Biological Signaling, Scientific Communication, Systems Biology, Research Excellence.

Introduction

Modern biological research increasingly recognizes communication and information exchange as fundamental components of living systems. Guenther Witzany has contributed to this perspective by examining the role of signaling processes across organisms and biological structures. His work bridges scientific and philosophical approaches, offering frameworks that help explain regulatory interactions within genetics, cellular systems, and evolutionary processes. Such interdisciplinary scholarship contributes to broader understanding of biological complexity and adaptive behavior.[3]

Research Profile

Guenther Witzany’s research profile reflects expertise in molecular biology, genetics, evolutionary studies, and theoretical biology. His scholarly activities emphasize communication-based interpretations of biological processes and the significance of information exchange in living organisms. With a documented publication record and international citations, his work contributes to academic discussions regarding the mechanisms through which biological systems coordinate, regulate, and evolve.[1]

Research Contributions

A significant aspect of Witzany’s scholarship involves the development of biocommunication theory, which investigates how organisms employ signs, signals, and contextual interactions. His research highlights the communicative dimensions of cellular activity, genetic regulation, and evolutionary adaptation. These contributions have provided conceptual models used to interpret biological organization beyond purely mechanistic explanations, encouraging interdisciplinary dialogue among researchers from multiple scientific domains.[4]

Publications

  • Biocommunication and Natural Genome Editing.
  • Introduction to Biosemiotics: The New Biological Synthesis.
  • Biocommunication of Fungi.
  • Biocommunication of Plants.
  • Communication and Information Exchange in Evolutionary Systems.

Research Impact

The research impact of Guenther Witzany is reflected through citation performance, continued scholarly engagement, and the adoption of communication-centered perspectives within biological sciences. His publications have contributed to discussions regarding genome regulation, evolutionary innovation, and biological information processing. By encouraging integration between empirical research and conceptual analysis, his work has influenced interdisciplinary investigations across molecular biology and related scientific fields.[2]

Award Suitability

The Excellence in Research Award recognizes sustained scholarly achievement, publication quality, citation influence, and contributions to advancing scientific understanding. Guenther Witzany’s interdisciplinary research portfolio aligns with these criteria through his established record of publications, measurable research impact, and continued engagement with emerging questions in genetics, molecular biology, and evolutionary theory. His work demonstrates both academic rigor and relevance to contemporary scientific inquiry.[5]

Conclusion

Guenther Witzany’s research contributions illustrate the value of interdisciplinary approaches in understanding biological systems. Through investigations of communication, information exchange, and evolutionary processes, he has contributed to scientific discussions that extend across multiple domains of biology. His publication record, citation performance, and conceptual innovations support recognition through the Excellence in Research Award and highlight the continuing relevance of his scholarly work within contemporary life sciences.

References

  1. Elsevier. (n.d.). Scopus author details: Guenther Witzany, Author ID 14629725000. Scopus.
    https://www.scopus.com/authid/detail.uri?authorId=14629725000
  2. Witzany, G. (2016). The biocommunication method: On the road to an integrative biology. Taylor & Francis.
    https://doi.org/10.1080/19420889.2016.1164374
  3. Witzany, G. (2025). Plant Growth and Development from Biocommunication Perspective. MDPI.
    https://doi.org/10.3390/ijpb16020063
  4. Witzany, G. (2012). Introduction: Keylevels of Biocommunication of Ciliates. Springer.
    https://doi.org/10.1007/978-3-319-32211-7_1
  5. International Research Excellence and Best Paper Awards. (n.d.). Award Evaluation and Recognition Framework.
    https://bestpaperawards.com/

Frederic Harb | Biochemistry | Research Excellence Award

Research Excellence Award

Frederic Harb
University of Balamand, Lebanon

Frederic Harb
Affiliation University of Balamand
Country Lebanon
Scopus ID 55509977300
Documents 56
Citations 462
h-index 14
Subject Area Biochemistry
Event Best Paper Awards
ORCID 0000-0001-5882-1619

This academic article summarizes the scholarly profile, research activity, and scientific contributions of Frederic Harb of the University of Balamand. The article presents a concise overview of publication performance, citation indicators, research impact, and suitability for recognition through the Research Excellence Award program.[1]

Abstract

Frederic Harb is a researcher affiliated with the University of Balamand whose academic work is primarily associated with biochemistry and related biomedical investigations. Based on available bibliometric indicators, the researcher has produced 56 indexed documents, accumulated 462 citations, and achieved an h-index of 14. These metrics indicate sustained scholarly activity and measurable influence within the scientific literature. The profile demonstrates engagement in peer-reviewed research, interdisciplinary collaboration, and knowledge dissemination. This article provides a concise assessment of research achievements, publication performance, scientific impact, and relevance to the objectives of the Research Excellence Award.[1]

Keywords

Biochemistry, Research Excellence Award, Scientific Publications, Citation Impact, Academic Recognition, Scholarly Contributions, University of Balamand, Research Assessment.

Introduction

Research evaluation commonly considers publication productivity, citation influence, and scholarly engagement. Frederic Harb has developed a documented academic profile within biochemistry, contributing to scientific literature through peer-reviewed publications and collaborative investigations. Such indicators provide a foundation for assessing research quality and academic standing.[1]

Research Profile

The researcher is affiliated with the University of Balamand in Lebanon. Bibliometric records indicate 56 indexed publications, 462 citations, and an h-index of 14. These metrics reflect continuing participation in scholarly communication and demonstrate an established presence within the academic research community.[1]

Research Contributions

Research contributions have focused on biochemistry and related scientific disciplines. Published studies contribute to understanding biological mechanisms and biomedical applications. Through collaborative investigations and peer-reviewed dissemination, the researcher has supported the advancement of scientific knowledge and promoted evidence-based research practices.[2]

Publications

The publication record includes 56 indexed documents covering diverse aspects of biochemistry. These outputs have appeared in peer-reviewed scholarly venues and contribute to scientific discourse. Publication activity demonstrates consistency in research engagement and participation in internationally recognized academic communication channels.[1]

Research Impact

Citation indicators provide evidence of research visibility and scholarly influence. With 462 citations and an h-index of 14, the researcher’s work has been referenced by other academics, demonstrating relevance within the scientific community. These metrics indicate measurable impact and ongoing recognition of published findings.[1]

Award Suitability

The available academic indicators suggest suitability for consideration within research recognition programs. Publication productivity, citation performance, and demonstrated scholarly engagement align with common evaluation criteria used by academic award committees when assessing scientific achievement and contribution to disciplinary advancement.[3]

Conclusion

Frederic Harb has established a documented research profile characterized by peer-reviewed publications, citation impact, and scientific engagement. The available evidence supports recognition of sustained academic contributions and highlights the researcher’s role in advancing biochemistry-related scholarship through scholarly communication and collaborative research activities.

References

  1. Elsevier. (n.d.). Scopus author details: Frederic Harb, Author ID 55509977300. Scopus.
    https://www.scopus.com/authid/detail.uri?authorId=55509977300
  2. ORCID. (n.d.). Research activities and scholarly profile of Frederic Harb.
    https://orcid.org/0000-0001-5882-1619
  3. Assessing Drug Administration Techniques in Zebrafish Models of Neurological Disease.
    https://www.mdpi.com/1422-0067/24/19/14898

  4. Mechanisms, Biomarkers, and Treatment Approaches for Diabetic Kidney Disease: Current Insights and Future Perspectives.
    https://www.mdpi.com/2077-0383/14/3/727

P. V. G. K Sarma | Biochemistry | Editorial Board Member

Assoc. Prof. Dr. P. V. G. K Sarma | Biochemistry | Editorial Board Member 

Associate Professor | Sri Venkateswara Institute of Medical Sciences | India

P. V. G. K. Sarma is a senior Indian biotechnologist and academic leader currently serving as the Head of the Department of Biotechnology at the Sri Venkateswara Institute of Medical Sciences, Tirupati, a position he has held since 2004, with his academic training from JNTU Hyderabad and a broad research portfolio reflected in over a hundred scholarly publications indexed across major journals. P. V. G. K. Sarma His core research spans genetic engineering, microbial biotechnology, functional genomics, structural bioinformatics, and stem cell differentiation with a strong translational focus on regenerative medicine and human disease. A major strength of his work lies in the in vitro differentiation of human CD34+ hematopoietic stem cells into diverse lineages including pancreatic β-cells, erythrocytes, podocytes, type-II pneumocytes, keratinocytes, chondrocytes, and megakaryocytes, contributing to the understanding of cellular plasticity and therapeutic cell generation. Parallel to this, he has made significant contributions to microbial pathogenesis, particularly on Staphylococcus aureus, elucidating molecular mechanisms of biofilm formation, metabolic regulation, and antimicrobial resistance, including MRSA, through gene expression, enzyme activity, and structure-based drug design approaches. His interdisciplinary research also addresses clinical and molecular genetics, with notable studies on mutations linked to congenital erythrocytosis, breast cancer, leukemia drug resistance, rheumatoid arthritis, tuberculosis susceptibility, cardiovascular disease, and pain genetics. During the COVID-19 period, his in silico investigations on antiviral compounds targeting SARS-CoV-2 proteins further demonstrated his adaptability across emerging biomedical challenges. He has consistently integrated wet-lab experimentation with computational biology, signaling pathways, and nanobiotechnology, including cytotoxicity studies of silver nanoparticles on human stem cells. Beyond research, he is actively involved in peer review for reputed international journals and contributes to clinical translational science through collaborations across medicine, genetics, and biotechnology. Collectively, his career reflects sustained leadership in Indian biomedical research, with impactful contributions to stem cell biology, microbial biotechnology, and molecular genetics aimed at advancing diagnostics, therapeutics, and regenerative strategies.

Profile: ORCID

Featured Publications

  1. Chandrasekhar, C., Kumar, P. S., & Sarma, P. V. G. K. (2019). Novel mutations in the kinase domain of BCR-ABL gene causing imatinib resistance in chronic myeloid leukemia patients. Scientific Reports, 9, 38672.

  2. Loganathan, S. E., Kattaru, S., Kodavala, S., Chandrasekhar, C., & Sarma, P. V. G. K. (2024). Prominent expression of COL2A1, ACAN and IHH genes observed in the differentiation of human hematopoietic stem cells into articular type of chondrocytes. Stem Cell Reviews and Reports, 20.

  3. Kodavala, S., Loganathan, S. E., Kattaru, S., Chandrasekhar, C., & Sarma, P. V. G. K. (2024). In vitro generation of epidermal keratinocytes from human CD34-positive hematopoietic stem cells. In Vitro Cellular & Developmental Biology – Animal, 60.

  4. Suthi, S., Mounika, A., & Sarma, P. V. G. K. (2023). Elevated acetate kinase (ackA) gene expression, activity, and biofilm formation observed in methicillin-resistant strains of Staphylococcus aureus. Journal of Genetic Engineering and Biotechnology, 21.

  5. Kattaru, S., Manne Mudhu, S., Loganathan, S. E., Kodavala, S., & Sarma, P. V. G. K. (2021). Increased insulin and GLUT2 gene expression and elevated glucokinase activity in β-like cells of islets of Langerhans differentiated from human haematopoietic stem cells on treatment with Costus igneus leaf extract. Molecular Biology Reports, 48.

P. V. G. K. Sarma has advanced translational biotechnology through pioneering stem cell differentiation, molecular genetics, and microbial pathogenesis research that directly supports regenerative medicine and precision diagnostics. His work bridges laboratory science with clinical and industrial applications, contributing to improved disease understanding, therapeutic development, and global biomedical innovation.

Ms. Shahrzad Manavi Nameghi | Biochemistry | Best Researcher Award

Ms. Shahrzad Manavi Nameghi | Biochemistry | Best Researcher Award

Researcher | Islamic Azad University Tehran Medical Sciences | Iran

Shahrzad Manavi Nameghi is an accomplished molecular genetics researcher from Tehran, Iran, recognized for her work in diabetes, cancer pathways, gene therapy, and stem cell research. She completed her M.Sc. in Molecular Biology-Genetics at Islamic Azad University, Tehran Medical Sciences in 2019, where her thesis focused on the relation between the GIPR gene and type 2 diabetes, following a B.Sc. in Cellular and Molecular Biology-Genetics from Shahid Bahonar University, Kerman in 2017. Professionally, she has served as a lab technician and R&D assistant at Khazar Lab, Tehran, and as a main lab technician at Mendel Medical Genetics Laboratory, contributing to projects involving DNA and RNA extraction, real-time PCR, pyro-sequencing, ARMS-PCR, MLPA-PCR, and other molecular techniques. She has also worked as a research assistant at the Endocrine and Metabolism Research Institute, gaining expertise in RFLP-PCR and sequencing analysis. Her research interests include molecular genetics, diabetes and cancer-related gene pathways, gene therapy, and stem cell therapy, and she is proficient in analytical techniques such as ELISA, fragment analysis, primer design, and basic R programming. Shahrzad has published multiple peer-reviewed articles in indexed journals including Gene Reports, Endocrine and Metabolic Science, and Regenerative Biology and Medicine, with a growing citation count of 15 and h-index of 2, and serves as an editor and peer reviewer for biomedical journals. She has presented her work at international conferences, including the IDF Congress in Korea and Brussels, and completed various certifications and workshops in peer review, manuscript preparation, laboratory techniques, and molecular biology. Her achievements, dedication to research, and involvement in scientific communities underscore her leadership potential and commitment to advancing biomedical science. For her significant contributions to molecular genetics, diabetes, and cancer research, and her active role in knowledge dissemination and mentorship, Shahrzad Manavi Nameghi is a highly deserving candidate for the Best Researcher Award.

Profiles: Google Scholar | ORCID | Scopus | LinkedIn | ResearchGate

Featured Publications

  1. Manavi Nameghi, S. (2024). Unraveling the molecular genetic basis of type 2 diabetes. Journal of Gene Reports. CiteScore: 3.3. Citations: 4. H-index: 2.

  2. Manavi Nameghi, S. (2024). Exploring the recent advancement and future prospects of personalized medicine in type 2 diabetes. Journal of Endocrine and Metabolic Science. CiteScore: 2.8.

  3. Manavi Nameghi, S. (2023). Cancer stem cells as a considerable barrier in anti-cancer treatments. Journal of Regenerative Biology and Medicine. CiteScore: 7.

  4. Manavi Nameghi, S. (2023). Association of GIPR gene variant on the risk of type 2 diabetes: A case control study. Journal of Endocrine and Metabolic Science. CiteScore: 2.8.

  5. Manavi Nameghi, S. (2022). Investigating the highlights of pluripotent stem cells in type 2 diabetes. Journal of Regenerative Biology and Medicine. CiteScore: 7.